Lmo2 transcription factor

2020-02-29 11:13 LMO2 is a member of a transcription factor family of proteins characterized by their cysteinerich, zincbinding LIM domains. Its expression is required early in hematopoiesis, and its deregulation leads to T

The transcription factors Scl and Lmo2 are crucial for development of all blood. lmo2 transcription factor

LMO2 is a component of multisubunit DNAbinding transcription factor complexes that regulate gene expression in hematopoietic stem and progenitor cell development. Enforced expression of LMO2 causes leukemia by inducing hematopoietic stem celllike features in Tcell progenitor cells, but the biochemical mechanisms of LMO2 function have not been fully elucidated.

Data show that the Lim domain only 2 (LMO2) regulatory element (element25) region consists of transcription factor GATA2binding myeloid ennhancer and RUNX proteinbinding Tcell repressor. Cryptic chromosomal rearrangements in the LMO2 gene lmo2 transcription factor

The LIMonly transcription factor LMO2 determines tumorigenic and angiogenic traits in glioma stem cells. S. H. Kim, E. J. Kim, M. Hitomi, S. Y. Oh, The LIMonly transcription factor LMO2 determines tumorigenic and angiogenic traits in glioma stem cells. In: How can the answer be improved? The transcription factors Scl and Lmo2 are crucial for development of all blood. An important early requirement for Scl in endothelial development has also been revealed recently in zebrafish embryos, supporting previous findings in scl embryoid bodies. lmo2 transcription factor The LMO2 gene encodes a transcription factor that is vital for angiogenesis and is highly expressed in DLBCLs that arise from the germinal center. A number of these miRNAs appear to be important in sustaining the clinical features of normal B cells and lymphomas. LMO2 is a member of a transcription factor family of proteins characterized by their cysteinerich, zincbinding LIM domains. 1 Its expression is required early in hematopoiesis, 2 and its deregulation leads to Tcell leukemias resulting from chromosomal translocations and insertional mutations. 3, 4 LMO2 expression is a strong predictor of

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